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CLS3374

Corning® HTS Transwell® 96 well permeable supports

HTS Transwell-96 Permeable Support with 8.0 μm pore polyester membrane, TC-treated, sterile, 2/cs

Synonym(s):

corning transwell, transwell corning

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About This Item

UNSPSC Code:
41121800
NACRES:
NB.13

material

flat bottom wells clear
polyethylene membrane
polystyrene

sterility

sterile

feature

lid
skirt
 plate format: 96 well

packaging

pack of 1
case of 2

manufacturer/tradename

Corning 3374

insert size

0.08 mL

membrane size

4.26 mm

membrane thickness

10.0 μm

size

96 wells

surface area

0.14 cm2 , cell growth area

well working volume

0.235 mL

color

clear plate

pore size

8.0 μm pore size

binding type

Tissue Culture (TC)-treated surface

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General description

  • HTS Transwell®-96 permeable supports have an array of 96 wells with permeable inserts connected by a rigid robotics-friendly tray
  • Product includes HTS Transwell®-96 permeable support insert, receiver plate (96 wells for growth or assay steps) and 1 sterile lid; 2 sets percase
  • 8.0 μm pores size PET (polyester) membrane
  • Membrane area for cell growth or cell migration is 0.143 cm² which is 20-50% greater than other brands
  • Large apical and basolateral access ports
  • Automation optimized design with multi-channel feeder ports, improved gripping surface and bar coding
  • All membranes are treated for cell attachment
  • Receiver plates are untreated
  • Lid – minimizes evaporation and protects against contamination

Legal Information

Corning is a registered trademark of Corning, Inc.
Transwell is a registered trademark of Corning, Inc.

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Xiang Jin et al.
Oncology letters, 18(2), 1218-1224 (2019-08-20)
Cervical carcinoma high-expressed long non-coding RNA (lncRNA) 1 (lncRNA-CCHE1) serves an oncogenic role in cervical and liver cancer. The present study aimed to explore the role of CCHE1 in pancreatic adenocarcinoma. CCHE1 expression was detected by reverse transcription-quantitative polymerase chain

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