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860679L

Avanti

C24:1 Ceramide-d7 (d18:1-d7/24:1)

Avanti Polar Lipids 860679L-1MG860746L-1MG

Synonym(s):

N-nervonoyl-D-erythro-sphingosine (d7)

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About This Item

Empirical Formula (Hill Notation):
C42H74D7NO3
CAS Number:
1840942-16-6
Molecular Weight:
655.14
UNSPSC Code:
12352100

Assay

>99% (TLC)

form

solution

packaging

package of 1 × 1 mg

manufacturer/tradename

Avanti Polar Lipids 860679L-1MG
860746L-1MG

concentration

1 mg/mL

shipped in

dry ice

storage temp.

−20°C

Packaging

2 mL Amber Glass Sealed Ampule (860679L-1MG)

Legal Information

Avanti Research is a trademark of Avanti Polar Lipids, LLC

Signal Word

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 2 - Skin Irrit. 2 - STOT SE 1 - STOT SE 3

Target Organs

Central nervous system

Storage Class Code

3 - Flammable liquids

WGK

WGK 2

Flash Point(F)

49.5 °F

Flash Point(C)

9.7 °C

Regulatory Listings

Regulatory Listings are mainly provided for chemical products. Only limited information can be provided here for non-chemical products. No entry means none of the components are listed. It is the user’s obligation to ensure the safe and legal use of the product.

EU REACH Annex XVII (Restriction List)

CAS No.

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Tejia Zhang et al.
Scientific reports, 9(1), 19939-19939 (2019-12-29)
Extensive characterisations of the zebrafish genome and proteome have established a foundation for the use of the zebrafish as a model organism; however, characterisation of the zebrafish lipidome has not been as comprehensive. In an effort to expand current knowledge
Hady Razak Hady et al.
Journal of clinical medicine, 8(12) (2019-12-18)
The liver plays a central role in the glucose and lipid metabolism. Studies performed on animal models have shown an important role of lipid accumulation in the induction of insulin resistance. We sought to explain whether in obese humans, the

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